Of which nobody knows what influence it had.
Yep
Mine too
I still get it.
Good point.
Or a receptor disfunctious so the cells can not work properly. I think in my case the a7nAchR plays a big rule. More so since I know this...
Ignore the disease process and everything will work like in healthy people.
Irresponsible
I said my ME pain is different from injury pain.
Cell adhesion molecules BMJ. 1999 Aug 7; 319(7206): 332–334. doi: 10.1136/bmj.319.7206.332 https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1126974/
I love this.
Right.
Depends on the definition of an organ. The skin is long accepted to be an organ. Now part of that organ is called an organ.
It could be the anwser to the mystery I could not explain with what is the textbooks.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3162128/ Positive allosteric modulators as an approach to nicotinic acetylcholine receptor- targeted...
More presice, a positive allosteric modulator. In other words , levamisole is a key for the a7nAChR backdoor.
I agree.
Only patient-reported scales were used and social network size information was not collected for healthy subjects.
The alpha7 nicotinic acetylcholine receptor as a pharmacological target for inflammation https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2042938/
Thus, vagus nerve and cholinergic agonists inhibit systemic inflammation by activating the noradrenergic splenic nerve via the alpha7nAChR...
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