"Mutations in IDO2 are common in the human population but it is unlikely that many would get ME/CFS. This is because the triggering is unlikely....
@Hutan @Rosie They are having a conference in NZ with Godlee speaking. https://ebmlive.org/ebm-events/
https://ebmlive.org/a-proposed-framework-for-the-pre-specification-of-statistical-analysis-methods-in-clinical-trials-pre-spec/ Meeting in Oxford...
This is arrogance on the part of the professionals. Why do they assume that people with ANY fatiguing illness aren't already functioning at their...
I'd be interested as well.
@Wonko Could you pm me also? Thanks
[ATTACH] I assume this means that the patient is always to blame if the " brilliant treatment " plan doesn't work. The quote is from Dr Cameron...
Conclusions Paediatricians need to be aware that physical activity patterns vary widely before recommending treatment. Duh! If I was a...
Do you mean that patients should supplement with glutamate if taking acetylcholinesterase inhibitors? I think galantamine was trialled about 25...
@mariovitali . Are you aware that Systrom is trialling pyridostigmine? He's looking at cardio effect, but is there any way of looking at liver effect?
I think there's a study of tVNS underway at Vanderbilt.
Just browsing Twitter thread for conference in my local university. Several presentations on this. Have you heard of it before in ME research?
@mariovitali Great news
Role of mast cells?
Cheney did suggest anti- gravitational exercise with a rebounder and some type of swing chair.
The Polish arm of Mortens study was looking at cryotherapy.
Having tried to follow CPET discussions, are they really of any value?
I liked your " break even point" as a way of explaining the very fine balance of activity
5. What role do bile acids play ?
3. Do Tuft cells play a role? 4. What about JAK inhibitors ( Lombardi)?
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