There are two issues here.
The first is whether the test is a highly accurate measure of the antibody titre in the individual. I am suggesting it isn't due to sampling and processing variability.
The second is whether the level of circulating antibodies will help prevent infection during the...
The FDA advice is correct, the tests do not indicate how "protected" you are. The tests are not reliable enough for any sort of quantitative prediction about "protection". The test shows that you have seroconverted and that is all that matters as far as the test goes.
Convenience studies like this don't provide direct evidence about cause-effect relationships like this, yet that is what they are trying to suggest in the conclusion...
It's funny how patients are never included as part of the Delphi method review group.
No long-covid patients want this to be called "Post COVID Syndrome".
One key problem with their criteria is that it poorly discriminates between individuals who have pulmonary damage and those who have other...
@hope123
Additionally, I partially identify with the "Maria" case study (on a disability pension, no partner or kids), although I am much younger and have been ill since I was a teenager (and I have a different pattern of symptoms).
I can tell you that the proposed interventions for Maria, are...
The problem is that depression (or anxiety) are symptoms, not causes.
The underlying causes are the lack of satisfaction of human needs, but few in the field (researchers or clinicians) bother to actually map the relationships between human need and satisfiers or lack thereof.
I think there...
This is from a month ago, but a prospective study of wet markets in Wuhan in 2019 found no evidence of bats or pangolins being sold.
https://pubmed.ncbi.nlm.nih.gov/34099828/
Actually, I was wrong, they have similar names, but TTP is apparently a different syndrome (despite having the same set of symptoms), and is associated with a different autoantibody (against ADAMTS13).
There is a very rare genetic cause, but it only explains 1% of TTP cases, the rest are...
This might be a disaster. Heparin sulphate does indeed bind to the spike protein, which I have previously mentioned, but I have proposed a similar mechanism for the thrombosis with thrombocytopenia syndrome, due to B-cell cocapture of PF4-Heparan sulfate-spike protein complexes.
Life here has been mostly normal, besides mandatory check-ins at retail premises.
Many people in Australia are yearning for the time before (ASAP) and seem to mistakenly think that is the situation in the UK.
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