It is correct to say they have not proven causation, just association, and statistically limited association at that due to the sample size. Like @Valentijn I am interested in this in comparison to repeat CPET findings. There is no question this study needs replication on a larger data set...
Actually it goes back to the 1930s at least. Its just the the term biopsychosocial was proposed in 77. Before then it had other names, and was proposed again and again. It was Engel's paper that took off, mostly because he framed it about medicine in general and not psychogenic psychiatry.
I did it for years. Here is what happened. I became massively sleep deprived. Then my OI went nuts and I started passing out and falling down all over the place. Sleep hygiene might be good for mild insomnia, but its not reliable for severe circadian issues.
I have done the same thing, and probably will again. I don't like the sound of "one" and usually substitute "you" but the context does not always disambiguate the meaning.
This is not just psychiatry, its also the other doctors who pass the patients on to psychiatry. Doctors often call patients like this "heartsink" patients, because they cannot help and they feel bad about it. How they handle this is however an issue in medical training ... failed training...
I think the risk of diabetes and CVD is probably increased. I think the profile is different though. I think ME can look like mild diabetes if there is enough of a problem with sugar metabolism in ME. However this will increase insulin, and hence insulin resistance, over time. I suspect that...
That is my current interpretation. They have moved away from objective outcome measures when they found that all of them reliably discredit their conclusions.
I think it was Peter White who made the claim that objective measures are not that important, if I recall correctly. He was defending using subjective outcomes in PACE.
I think the entire notion of mind is beyond its use by date. There is so much fallacious and baseless discussion of mind. Discussing brain, brain function, and learned experience avoids many of the logical pitfalls involved with discussing mind.
I have long suspected this might be due not just to disregulation of sleep, though we only just discovered the second sleep regulation area in the brainstem. I suspect we have issues with insufficient PGD2, which is the hormone the brain uses to tell most of the brain cells to go quiet. If you...
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