Webinar: COVID-19 as a Trigger for ME/CFS: Severity Biomarkers and Underlying Mechanisms with Dr Carmen Scheibenbogen

[Sly Saint]

October 24 @ 10:00 am - 11:00 am PDT
Organizer Solve M.E.

Since the beginning of the pandemic, SARS-CoV-2 has been suspected of causing chronic fatigue syndrome (ME/CFS). A well-controlled cohort study conducted by a group of researchers from Charité – Universitätsmedizin Berlin and the Max Delbrück Center for Molecular Medicine (MDC) has now shown that in the subset of patients who suffer from fatigue and exertion intolerance after mild-COVID-19, around half of patients meet the diagnostic criteria for ME/CFS.

Their findings also describe differences between post-COVID ME/CFS patients and ME/CFS patients in the correlation between hand grip strength and inflammatory biomarkers. This may indicate differences in underlying mechanisms. The researchers’ findings have been published in Nature Communications.

In this webinar, Prof. Dr. Carmen Scheibenbogen (Acting Director of Charité’s Institute of Medical Immunology on Campus Virchow-Klinikum and Solve M.E. Ramsay Research Grant awardee) will discuss the findings of this study, and how the new findings may help researchers to develop specific treatments for post-Covid ME/CFS and ME/CFS.

Register here.

https://solvecfs.org/event/covid-19...everity-biomarkers-and-underlying-mechanisms/

 

[BrightCandle]

This may indicate differences in underlying mechanisms.

This is what we are afraid of when the various ME/CFS communities go about promoting Long Covid and not being concerned about the funding gap, if something helps Long Haulers and not ME patients we will been dumped by our own advocacy groups for years. Its likely they are similar and progress in one helps the other but its by no means guaranteed especially if the solution involves antivirals against Covid or the spike protein.

 

[Peter T]

We do not yet have an operational clinical definition of Long Covid and on the basis of current knowledge Long Covid is even more likely to a heterogeneous grouping than ME.

It would only currently be meaningful to say Long Covid and ME diverge if the comparisons are made between people with Long Covid who also meet the clinical definition of ME and people with ME who have been shown not to have had any active Covid 19 infection.

ME is triggered by lots of different viruses and it would be unreasonable to claim Covid 19 never triggers ME, rather it remains to see what percentage of those with long term sequelae following Covid have ME and what percentage do not. Then do people with Covid triggered ME also have some of the other symptoms following Covid experienced by other people with long term Covid sequelae who don’t meet the diagnosis for ME or not?

 

[Sly Saint]

COVID-19 as a Trigger for ME/CFS: Severity Biomarkers and Underlying Mechanisms

 

[Mij]

"Therapy studies to remove autoantibodies are urgently needed, the first results of our immunoadsorption study at ME/CFS show that many people feel better afterwards. We are preparing studies with drugs for B-cell depletion, but unfortunately everything takes so long"
https://twitter.com/user/status/1853052432202248515