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Yes but even in this area it does not look impressive. The RECOVER study on ME/CFS post-covid did not include medical examinations so it was worse than the EBV studies we have.

Isn't that what happened in the US with the Long Covid money?

Quote from the retraction watch article: Back in 2019, we made an overview of guidelines and policies that referred to the GET review here: https://www.s4me.info/threads/the-influence-of-the-cochrane-review-on-get.11768/

Where did this funding come from? Was it a government grant that they lost or simply overall budget cuts?

Any articles on this because it seems like a strange decision.

Yes GRADE recommends downgrading for imprecision if the CI crosses the threshold of interest. Which is the case here.

I noticed that Lillebeth Larun has now responded to a comment: https://www.cochranelibrary.com/cdsr/doi/10.1002/14651858.CD003200.pub9/detailed-comment/en?messageId=446137675

Thanks for all the comments and suggestions. We have just posted the following poll on social media based on 3 options that we like the best thus far. Which name should we choose for our account and blog? 1) ME/CFS Science 2) ME/CFS In Depth 3) Science unravelled 4) None of the above (keep...

https://eprints.whiterose.ac.uk/164981/1/20200428ConversiondisordermisdagnosisGHPaccepted_combined_1_.pdf

Thanks. The data is in .raw filetype and have some trouble converting it to something like CSV or Excel. Anyone who has more experience in this?

This study is listed on MapME/CFS but it doesn't include the raw data from what I can see, only summary statistics of their analyses and modelling.

Just posting the main results here: The Japanese used next-generation sequencing (NGS) and got the following results: They also found that it was related to an infectious-onset and shorter illness duration (perhaps that explains why Ryback only seem to find it in the mild/moderate but not...

Any ideas at what this may point to? If it is not specific antigen driven expansion, what else might be causing this? And why would B-cell abnormalities make sense, as Nath said, if it is likely not related to a specific antigen drive?

Wrote a short section about this in our 2021 review of ME/CFS studies:

Thanks, the two studies these refer to are I believe: 2020 Lipkin study: Plasma proteomic profiling suggests an association between antigen driven clonal B cell expansion and ME/CFS | PLOS ONE 2021 Sato study: Skewing of the B cell receptor repertoire in myalgic encephalomyelitis/chronic...

Looks interesting. The first author is Audrey Ryback who is also trying to replicate the 'something in the blood' theory, as explained by Simon Mcgrath here: Remarkable researchers hunting for ‘something in the blood’ of people with ME | Science for ME

More than a month later and there is still no peer review or reply from the authors to the comments made.

Thanks for the suggestions. It seems that misuse of the term skeptic has become so common (i.e. as in climate skeptics) that a correct use of the term has become confusing to most people. We still plan on changing the name but needed some more time to consider the best options. Will probably...

Later in the text they write: So that suggests that one ME/CFS symptom is enough to meet the ME/CFS-like definition. What they call ME/CFS should have been named ME/CFS-like to be consistent with the literature. And what they named ME/CFS-like shouldn't have been named anything.

Bit sad that they got more than 1 billion in funding and this is the best they could do...