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I meant to say the observed pattern, it seems my attention to detail is poor today.

Notably, the assumption is the opposite direction of Phair's metabolic trap hypothesis.

I am really surprised as to why journalists and scientists aren't bothering to look whether the same thing happens after other infections and vaccinations. The observed pattern in antibody titres is typical post-infection and not yet a cause for concern.

Depression was found in 15.6% of CFS patients, which does not seem like an unreasonable estimate. Cancer prevalence was 6.4% in CFS patients, which seems high, given a rate of 1.9% in age matched controls. In principle, there shouldn't be a presentation bias for cancer in CFS patients, unless...

Cortisol is a feed-forward metabolic hormone, it's purpose is to provide sufficient blood glucose upon wakening and smooth the blood glucose level over the day given anticipated activity demands (insulin is a feed-back metabolic hormone...). Despite all the pop-psychology on the contrary, it's...

There is one major difference: New Zealand (and Australia until recently) had very little community transmission, which means there is a very different demographic distribution as to who was infected. We cannot and should not assume that testing is somehow a magnitude of order lower than it...

People like SW often retweet things they haven't actually read...

I'm not anywhere near as optimistic as that. Nothing is going to change until we have some understanding of the pathology.

Wait, I got it: Magic! Someone must have cast a hex on them! No wait that's not it how about: They displeased the gods! Perhaps they were nasty people in a past life and have to atone in this one?

Note how all of the references for "enhanced sympathetic activity" are papers written by Wyller! He takes self-citation to a whole new level. Studies of adults have not found differences in levels of catecholamines compared to healthy controls. The concept of "sustained arousal" also...

I don't get too excited about results that largely overlap with healthy sedentary individuals like most of the cortisol studies I have seen. If it was a cause, it would already be considered a biomarker.

Why are they even at the hospital getting scans if they aren't sick. There have been several "asymptomatic" cases discussed recently. The first is an individual had shortness of breath but no fever, but was considered "asymptomatic" as the doctors claimed the shortness of breath and mild cough...

I've already addressed this nonsense about there being any significant proportion of individuals who magically have COVID without ever having any symptoms... (don't confuse reporting biases, test contamination, lack of test specificity or pre/post symptomatic cases with "asymptomatic" cases.)...

I guess the people at the Swedish Public Health Agency are really bad at arithmetic...

At that rate (per day), they'll achieve herd immunity in 4 years!

At the current rate, the USA is going to overtake Sweden, Spain, Italy and France in deaths per million in a matter of 2-3 months!

I'll put your name on my paper if you put my name on yours! ;)

https://www.nejm.org/doi/full/10.1056/NEJMoa2022483 Notable aspects: the highest dose (250 μg) caused headache in 100% of recipients, fever in over 50%, along with severe fatigue in some recipients of this dosage. One recipient in the lowest dosage (25 μg) group had a significant immunological...

It wouldn't say it is "quite common", but there are false positives, typically due to contamination. It's also possible that the first infection was SARS-2, it wasn't fully cleared and the second infection is something else.

It proves that no one outside of the MUS world has ever heard of the PACE trial...