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Let s4me allocate it over time (say a period of 10-15 years). I don't really have great hopes otherwise. LongCOVID has set things backwards rather than forwards.

Basically some people are recovering naturally, adjusting their activity levels naturally and these "rehabilitation specialists" are thinking they're making a difference when they're largely bystanders.

As previously discussed, in many patients, the symptoms are not the same, depending on the triggering activity. Four out of five people report differences: https://www.s4me.info/threads/poll-physical-vs-cognitive-pem-same-or-different.16948/ Cognitive activity does not cause any additional...

Note that Hoffman reflex is a muscular reflex (due to muscle spindle afferents). It is not a pain signal and some people are naturally hyper-reflexive. The effect size for the H-reflex rate-dependent depression wasn't super large and there was substantial overlap between patients and controls...

Because people like to oversimplfy things to the point of inaccuracy. Many people don't shake when anxious, it is separate learned behaviour and vasovagal syncope is largely is a consequence of hyperventilation. So it is cognition->behaviour->cerebral blood flow consequences->vasovagal...

This is largely untrue, at least with respect to training of muscles. You are greatly overstating the effect of cognition/emotions on autonomic regulation.

From the introduction: So they are training medical doctors to push non-consensus/non-evidence based explanations. How can you search for solutions if you have no understanding of the cause? It begs the question. So we come to the crux of it: This training isn't about patient care at...

It is not just DecodeME that gives us clues. I think PEM is woefully undercharacterised. The overall symptomatic patterns (eg pain associated with fatigue, exacerbation of OI or POTS, headaches etc) and durations is not always consistent, it varies based on task and duration - whether cognitive...

This was an issue for me, I had frequent infections and antibiotic treatments when I was young - and I contracted diseases like mumps despite being fully vaccinated.

If you're suffering from ongoing brain fog, you can't code to any level of quality. I suspect he simply recovered naturally and stated working on his project as his functionality improved.

CIDP is not Long Covid and has established diagnosis and treatment, although looking at stories on reddit /GBS, patients frequently seem to have delayed or misdiagnoses.

I can't remember, it was probably speculation.

That is ChatGPT slop in an nutshell. Note the Thesis states: I suspect the author doesn't have the greatest skills in written English.

This was published, not sure if any changes Pharmacological Blockade of Muscle Afferents and Perception of Effort: A Systematic Review with Meta-analysis Systematic Review Published: 01 November 2022 Volume 53, pages 415–435, (2023)...

Such awful construct validity!

A doozy. :D

Came across this and wanted to see if there was a discussion here. I'm not sure this is a dorsal root ganglion based autoimmune disorder. But the methods are novel (and I don't think screening of CSF antibodies using rat DRG is "idiocy", it's a common method to try and uncover otherwise unknown...

ME/CFS Zetetic

This finding might be common sense, but it certainly contradicts the whole "fatigue is a stop signal" nonsense.

I'm revisiting this I think the proposed pathogenic mechanisms "Possible pathophysiological mechanisms are central sensitization, moderately or intermittently increased cerebrospinal pressure, the particular characteristics of the blood–nerve barrier in the dorsal root ganglia, and axonal...