In the second post, I highlight how the term was used in a long Covid paper.
I tried to get to the bottom of their claims..
Your paper says this about clinophilia:
In addition, actigraphy showed a tendency towards high night-to-night variability, high inactivity index and relatively low inter-daily stability during the monitoring. These findings suggest clinophilia and poor sleep hygiene, which may contribute to and be affected by increased fatigue in a bidirectional relationship [
54].
Our results are consistent with the reports of 65 subjects 3 months after hospitalization due to COVID-19 infection, which showed a reduced inter-daily stability of 0.59 and a mean sleep efficiency of 84.6% [
55]. Clinophilia is a symptom usually associated with mood disorders [
43], and in our cohort, a large percentage (78.6%) of patients reported depressive symptoms.
54
says this about clinophilia:
Nevertheless, without this procedure [polysomnography], it is difficult to distinguish whether hypersomnia consists of actual extended sleep or whether it simply represents an extra time spent in bed without necessarily sleeping, known as clinophilia.
(…)
Taken together, there is no objective evidence supporting the view that patients with mood disorder have either abnormal mean sleep latency on the MSLT or objective extended nocturnal sleep. However, these patients spent a substantial amount of time in bed, acknowledged as ‘resting’ more than ‘sleeping’ (called clinophilia), with major distress and impacts on the natural course of mood disorders.
I can’t find any support for the first paper’s claims that clinophilia contributes to increased fatigue. The other way around would be plausible, although there’s no support for this in the sections that directly mention clinophilia.
Before the second paragraph, they mention a study with reference 57.
57 in 54 is
paywalled, but it tries to distinguish primary and psychiatric hypersomnia (excessive sleepiness):
The aim of this study was to assess whether polysomnography aids in the differential diagnosis of these two disorders.
Our findings indicate that psychiatric hypersomnia is a disorder of hyperarousal, whereas primary hypersomnia is a disorder of hypoarousal.
Based on the abstract, there is nothing in the design or conclusion of 57 that makes it suitable to determine if clinophilia causes ‘major distress and impacts on the natural course of mood disorders’.
54 (in the first paper) also mentions some other studies on bipolar depression and major depressive disorder, but I’m not sure they are relevant.
43 in the first paper is paywalled, but the claim that clinophilia is usually associated with mood disorders is imprecise at best. My guess is that clinophilia is associated with any severe illness, as sick people tend to want to rest more than healthy people. The reason that clinophilia doesn’t show up in biomedical research is that they don’t have a freudian fetish.
In summary, it seems like your original paper has taken some creative liberties..