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what is the classic (medical) method to have this osmose stress measured ? (so, for ppl without nano needle) @Jonathan Edwards walking at altitude ? diving ?

most interesting may be indeed the "psychology" behind this odd publication. surely, they wouldnt do anything that sheds a bad light on "stanford".

@B_V you have ptosis on the RIGHT eye and inflammation only in one leg, the LEFT tigh ? so, only the first (stanford) left leg biopsy showed inflammation ? would have 1,000 questions, but what exactly were your eye problems when you saw the doctor and the ptosis was diagnosed ? and what...

where is the signal for MDD major depression disorder ? compare it with mecfs.

ok, they will have checked if ppl with e.g. less blood cell deformability dont have the same signal. (more diseases show less deformability faik) if they say, it detects "mecfs" then other diseases must have a different signal. perhaps there is a signal-comparison with other diseases in the study.

they must be sure that this impedence problem is different with autism, MS, fibromyalgia (...) ? they cant have just controlled with healthy people ?

it doesnt have to do with the sodium inhibitor pump, did they mean to hint that in the paper ?

i understand, pwmecfs PBMC perform badly under (hyper)osmotic stress

Actinobacterium Eggerthella enzyme strain CGR2 A. Eggerthella strain cgr2 from the gut deactivates Quabain. Eggerthella cgr2 may have benefits for ppl with high quabain, even though the bacterium is considered pathogenic ? endogenous ouabain (EO ) and related substances can be produced in the...

Oral fluoroquinolones and risk of fibromyalgia ... not found Received 22 March 2018; Revised 26 August 2018; Accepted 8 September 2018 Correspondence Mahyar Etminan, Assistant Professor of Ophthalmology and Visual Sciences, Faculty of Medicine, Department of Ophthalmology and Visual...

@MaximilianKohler perhaps you can look into that ? Actinobacterium Eggerthella enzyme strain CGR2 only 40% of the population may have this strain. only this one deactivates ouabain. but then sodium in blood cells increases, sodium pump is shut down (problem in pwMecfs). we may not produce...

Nano-Gold and Silver work - since they ride on Exosomes ? Exosomes guard the Crime Scene ..but why do they leave after 96h ?

the women disappeared ?

we may produce endogenous Ouabain in the Hypothalamus if this is caused by such a process of endogenous production, then an antagonist could be Rostafuroxin (german wiki again only) ? .. am just wondering, since so much has been tried already... perhaps this one too ?

sorry for the mess, all quotes no link: Exosomes — packed with RNA, DNA, and proteins microRNAs — short stretches of RNA — Removing a specific microRNA molecule — miR-150 – eased disease severity, inflammation, and loss of myelin in a mouse model of multiple sclerosis (MS), researchers...

it may be because i have no research/medical understanding... i cant believe, its not known what it is. actually, i feel arsed when i understand right... its been shown already years ago, that pretty much everyone (ill) has "something in the blood". in MS they found miRNA150 which comes on...

would this fluge/mella "oxygen/lactate issue" be shown in the usual lactate blood test after exercise (taking a drop of blood from the finger tip) ? (perhaps it may show in the mecfs case some "unusual" result ?)

fluge & co said (i think): increased oxygene consumption AND increased lactate production is it known if in the healthy blood "infection" the lactate production increased, too ?

these "tests" dont seem overly complicated to me. the implications of the signalling seem important. so much is going "wrong".

what symptoms may the signal-normalization fix in mecfs ? muscles ? brain fog ? also, would be interesting to know if the MS patients have a similar signal deviation ? since copaxone helps them. does copaxone in MS help for the same reason (signal improvement) ?