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We have a thread on the Lightning Process here 'Lightning Process' which links to other threads and resources.

There's some discussion about an article 'Far too much advertising is being done for Long Covid' on the website of Syp Wynia, 'Wynia's Week' posted by @MatthiasRiem here: BPS attempts at psychologizing #LongCovid That 'a Dutch youth magazine for recreational mathematics' exists says something...

I've written a letter of complaint - emails can be sent to the editor of the Life Style section - Emma.chamberlain@stuff.co.nz. It's nice to see the ANZMES website does not endorse the Lightning Process - their comment is quoted in the article. At one point the article suggests LP can make...

Fiona Fraser is the writer of the article and Jenny Oliver is the LP practitioner - basically it's an ad. It's just nonsense.

I haven't checked out the study but people interested in insulin resistance and Type 2 diabetes might want to check out this recent NZ study:

Yeah, grim BBC series, about a unit uncovering police corruption, and, corruption in general. I've yet to see the final episode of this latest series, but typically there's some minor wins with a lot of collateral damage, and the institutionalised corruption grinds on. Those who stand against...

That high diagnostic stability isn't something to crow about when even progressive supranuclear palsy doesn't change a functional neurological disorder label. I mean, at what point in the next few years is this poor woman allowed to actually be 100% ill, rather than having symptoms that she...

Not much left for it to 'secondarily revolve around', I would have thought, except perhaps 'time', which is probably the factor doing the heavy lifting.

While I appreciate Sonya asking for the information, I do wonder about the reason she gives - that people are anxiously speculating on social media and, it is inferred, are suffering as a result. There are much better reasons to be concerned. One is that a delay means that people, including...

I was assuming that a GWAS would produce data that could be used to identify the rare variants that do cause 'genetic' diseases- diseases pretty much directly caused by a genetic variant (along with, of course, its main purpose of identifying genetic variation that might create a vulnerability...

Russell Fleming is, I believe, the Content Manager for the ME Association and is a member here. Yes, it is an odd document, particularly given how well-informed Charles and Russell are and the usefulness of most of their advocacy. I'm not in the UK either, but I think commenting is valid...

Posts about the finding of epigenetic changes in lung cells that persist after Covid-19 'recovery' have been moved to The biology of coronavirus COVID-19 - including research and treatments

@Russell Fleming, I think there are important points in this thread. Could the MEA please urgently review its statement on exercise, particularly noting that there is no evidence that exercise, not even flexible, gentle exercise, cures or improves ME/CFS?

Split thread from Genome-wide association study identifies RNF123 locus as associated with chronic widespread musculoskeletal pain, Md Shafiqur Rahman, et al, 2020 *** I was reading about calcium channels the other day. This reference about them is very readable and interesting. Don't be put...

I guess the issue is 'useful for what'? Clearly, such treatments are useless for curing or even improving ME/CFS. But, say you are an elite athlete, and you've just had 6 months mostly lying in bed following Covid-19, with ME/CFS symptoms. Now, you are feeling better and want to start...

Authors also include Darren Brown and Michelle Bull. @PhysiosforME Yes, I agree with you. But, people will turn up at the exercise therapist's door, and if the exercise therapist knows to say what is on that little diagram (stop pushing through, rest, pace) and can explain the concept of...

I don't know. Maybe it's best to wait and see if the DecodeME effort comes up with anything (while using international research funds for other useful research in the meantime)? Can you say more about why another GWAS is important at this time @Snowdrop? Is your thinking that any GWAS takes...

I guess I was suggesting that it could be worth looking for stuff in particular. If there's just a handful of people with a particular pathogenic variation, maybe that would not show up as being significant in DecodeME. (?) But, for those particular people with the pathogenic variation...

Maybe a bit of research work that could be done parallel to DecodeME (the UK ME/CFS GWAS) would be to go through the literature and identify all the known gene mutations likely to be directly causing conditions with symptoms like ME/CFS - like this ENO3 variant. Then the DecodeME data could be...

@mariovitali started a thread to discuss glycogen storage diseases here: Glycogen Storage Diseases and ME/CFS