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This is just in the patient group with the abnormal %CBF change. The first sentence in that paragraph is a bit confusing - the %CBF reduction was not found to be correlated with all those factors. When they say 'related' they mean that they examined the relationship between %CBF and each of the...

I agree that us mentioning McEvedy and Beard in articles like this might well strengthen people's association between ME/CFS and psychosomatic illness. But this article was mostly good, and I appreciate that the health correspondent took the time to write about the issue. Jim White seems to...

1135 ME/CFS patients attended the clinic in ten years (met Fukuda and Carruthers(is that CCC) criteria) and who had a tilt test due to suspicion of orthostatic intolerance. 664 patients had a normal HR and BP response. Patients younger than 18 years or with a very high BMI were excluded...

Very small control group The study found a decent correlation between fibromyalgia severity and mitochondrial function. However, it didn't find a correlation between the symptoms severity scale and mitochondrial function. The SSS measures a mixed bag of symptoms, and so the lack of a...

The first figure B above does seem to suggest that most of the fibromyalgia samples are lower than the healthy control samples though. There are some possible reasons why faulty mitochondria could be the problem even though some of the fibromyalgia samples look the same as the control...

I expect so, I was trying to indicate the catchment region of the service but I am lacking in UK geography knowledge. I'll change it.

Sorry, my post was more questions than a statement. I need to emphasise there are lots of ifs and maybes with the idea that ME/CFS is brain damage. And the rapid remissions would seem to go against the idea. I just couldn't recall if we had seen the suggestion that EBV in the form of a...

This seems to be suggesting that EBV can cause brain damage. Perhaps supporting the hypothesis that ME/CFS is damage to a different part of the brain.

There are a couple of posts about the service here: Maeve Boothby O'Neill - articles about her life, death and inquest One poster says that the service is not taking patients who live outside Leeds.

We already have a thread for the service: (Not a recommendation) [UK] National Inpatient Centre for Psychological Medicine (NICPM) The side panel explainer 'What is ME?' looks to have been changed, if it previously talked about GET and CBT. Here is the current version:

Oculomotor Behaviour in Individuals with Long COVID-19, 2024, González-Vides et al. replication, also investigation of the impact of longer duration tasks, PEM, exercise, and investigation in ME/CFS cohort

It's such an interesting field. Here's a paper with some background. Individual differences in human eye movements: An oculomotor signature? 2017 It seems that an individual's specific combination of oculomotor measures is identifying and has good re-test reliability. Also that brain trauma and...

Interesting paper Oculomotor Behaviour in Individuals with Long COVID-19, 2024, González-Vides et al. There is some link with the 'Spanish Association of Persistent Covid'

From the abstract, this looks interesting - good sample size, good differentiation between the cohorts, looks like all of the measures are reported and all are significantly different and in the same direction. The controls are older (mean age 53.5 years versus 46.4 years in the LC cases)...

Such a good post @Ken Turnbull. I had to do a psychological assessment as part of a recruitment process and some of the questions asked were nonsense and for most, it was obvious what would be a 'wrong' answer, making gaming inevitable. After being offered the job, I mentioned that their...

It's an interesting theory. The previous paper is discussed here:HERV activation segregates ME/CFS from FM and defines a novel nosological entity for patients fulfilling both clinical criteria, 2023, Gimenez-Orenga Of course, a big problem with this study is the size of the samples e.g. only...

Figure 1: a is the responders, b is the non-responders. SF-36 PF

They don't say that SF-36 was the primary outcome. A lot (really a lot) of other things were measured too, e.g. Fatigue Severity Scale, Bell score, PEM-DSQ. It's looking as though the assessment point was actually 4 weeks after treatment. An assessment period that short is especially prone to...

Please can they blind these future studies? Two thirds of patients improving from 25 to 60 on the SF-36 PF score over 6 months in a small unblinded study is interesting, but still within the bounds of a placebo response.