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Why? I actually doubt it is genetic as well, but I am curious as to why you make this statement. It seems that almost once a month a new genetic marker is stumbled upon that suggests a form of channelopathy, so, I'm curious as to why you say this? Agreed, except I'm unclear how it works other...

Poor appetite? Neck pain? Who are these people?

Before they did anything, they should have performed the same metrics on HC and late stage Lyme (as opposed to early Lyme) and what they call PTLDS to see if there were any differences between LS Lyme and PTLDS. I suspect there would have been no meaningful differences. If that is the case...

I qualified for both the ME/CFS and Lyme parts of this study, and was in fact recruited to participate. Somewhere along the line it was decided I could not participate since I did in fact have both diseases. In a way, for me at least, that pretty much sums up the results.

Wisconsin is in the thick of things, too, when it comes to tick-borne diseases...

Is this just a case of suggesting the rooster makes the sun rise? Inference is no longer a strength of mine, but that deficit seems endemic in medicine so I don't feel too lonely.

"...ion handling..." A potential channelopathy reference again? Are we talking channelopathies as downstream effects, and abnormal RBC deformability simply further downstream? I know - way too speculative. Also, I always thought of channelopathies as the starting point, ie, genetic, usually...

Our's would be an acquired altered RBC deformability, if applicable - I think (it could be genetic, but adult onset in some and childhood onset in others makes that proposition seem less likely). This paper addresses hereditary hemolytic anemia. What about acquired hemolytic anemia, or...

This paper specifies skeletal muscle. Maybe the Australians were on to something with the calcium channelopathy theory. But the tie to EV escapes me. Are EVs remnants or blebs from skeletal muscle cells?

Not unlike the logic underpinning MS, truth be told.

Well, not obvious to everyone.

One of the authors is named Fukuda? Oh, sweet irony....

Very cool, in a not-cool-sort of way. I don't think they found a new tick. I think they found a species of babesia not normally found in Scotland. And one never found in sheep before, which is somewhat alarming. The article says the chances of contracting this form of babesia are remote. It was...

It is frightening just how many swaths of medicine not merely come up short, but suck. This is not what we were taught in the 60's and 70's and 80's - although we did start to learn in the 80's that dark, unexpected things lurked despite earlier medical posturing.

How to fail a given drug for a disease: trial it at insufficient dosages for too short a time frame. I wonder, though, what they imagine they are shotgunning mino at. All that voodoo entangled Tau Rastafarian biofilm Zen Protein comprised of...what? You know who got real good with this version...

Probably true, and I certainly enjoy reading that perspective. A corollary might be that suffering from a chronic disease is an exercise in learning that the medical community probably has no idea what they are on about - learning how doctors and researchers get things wrong. I mean no...

Clinicians and researchers tend to share the human trait of not seeing what they don't want to see. The NIH seems to enjoy an inexhaustible supply of this trait.

"Therefore our results represent a step forward for personality neuroscience within the study of biochemical systems and brain structures." Bets?

This phenomenon is not just restricted to HIV.

I'm glad I misunderstood, but not surprised; I'm almost getting used to misreading and mis-writing. Not quite, though.:unsure: