Introduction. After the acute form of COVID-19, 10 to 30% of patients tend to develop a post-covid syndrome or long-COVID-19. Research is actively being conducted into the causes of long-term COVID-19, one of which may be a violation of the immune response after SARS-CoV2 enters the body as a...
Molecular Mimicry Between Epstein-Barr Virus and Human Herpesvirus-6 Proteins and Central Nervous System Proteins: Implications for T and B Cell Immunogenicity in an In Silico Study.
preprint
Abstract
Background: The Epstein-Barr virus (EBV) and human herpesvirus 6 (HHV-6) are frequently...
Abstract
Aberrant immune responses to viral pathogens contribute to pathogenesis, but our understanding of pathological immune responses caused by viruses within the human virome, especially at a population scale, remains limited.
We analyzed whole-genome sequencing datasets of 6,321 Japanese...
Antibody responses to Epstein-Barr virus, human herpesvirus 6 and human herpesvirus 7 in patients with chronic fatigue syndrome
T Sairenji, K Yamanishi, Y Tachibana, G Bertoni, T Kurata
Abstract
To test for an association between chronic fatigue syndrome (CFS) and infections with Epstein-Barr...
Detection of herpes viruses in patients with myalgic encephalomyelitis /chronic fatigue syndrome in Belarus
Svetlana Orlova, Galina Rudjko, Olga Orlova, Tatsiana Dakukina
Abstract
Myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) is a multifactorial chronic disease. The etiology and...
Association of Active Human Herpesvirus-6, -7 and Parvovirus B19 Infection with Clinical Outcomes in Patients with Myalgic Encephalomyelitis/Chronic Fatigue Syndrome
Svetlana Chapenko, Angelika Krumina, Inara Logina, Santa Rasa, Maksims Chistjakovs, Alina Sultanova, Ludmila Viksna, Modra...
Full title:
From human herpes virus-6 reactivation to autoimmune reactivity against tight junctions and neuronal antigens, to inflammation, depression, and chronic fatigue syndrome due to Long COVID
Abstract
Inflammation and autoimmune responses contribute to the pathophysiology of Long COVID...
Latent virus reactivation in astronauts on the international space station
Mehta, Satish K.; Laudenslager, Mark L.; Stowe, Raymond P.; Crucian, Brian E.; Feiveson, Alan H.; Sams, Clarence F.; Pierson, Duane L.
Reactivation of latent herpes viruses was measured in 23 astronauts (18 male and 5...
HHV-6 encoded small non-coding RNAs define an intermediate and early stage in viral reactivation
Prusty BK, Gulve N, Chowdhury SR, Schuster M, Strempel S, Descamps V, Rudel T
Human herpesvirus 6A and 6B frequently acquires latency. HHV-6 activation has been associated with various human...
Abstract
Beyond their pulmonary disease, many COVID-19 patients experience a complex constellation of characteristics, including hyperinflammatory responses, autoimmune disorders, and coagulopathies. However, the pathogenesis of these aspects of COVID-19 is obscure. More than 90% of people are...
Full title: EBV/HHV-6A dUTPases contribute to Myalgic Encephalomyelitis/Chronic-Fatigue-Syndrome pathophysiology by enhancing TFH cell differentiation and extrafollicular activities
Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) is a chronic, debilitating multisystem illness of...
Abstract
Introduction: Chronic fatigue syndrome (CFS) is a neurological disease that is accompanied by excessive fatigue or tiredness. There are several reports confirming the association between Human Herpesvirus 6 (HHV-6) infection and CFS illness. This systematic review and meta-analysis...
Dr Ablashi is one of the discoverers of human herpesvirus 6.
http://microbeminded.com/2020/06/28/interview-with-dharam-ablashi-on-the-discovery-of-hhv6-and-its-involvement-in-chronic-inflammatory-disease/
by Philipp Schreiner, Thomas Harrer, Carmen Scheibenbogen, Stephanie Lamer, Andreas Schlosser, Robert K. Naviaux and Bhupesh K. Prusty
Published in ImmunoHorizons
Abstract
Myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) is a multifactorial disorder with many possible triggers...
Martin Lerner MD proposed that the reason why patients could have negative blood tests for virus yet still be infected was because the virus(es) that infected them became abortive and therefore unable to reproduce. What could cause this change?
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