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Or it could be certain individuals with vested interests using sockpuppet accounts? It wouldn't surprise me if people did this in the world of ME research and treatment to skew surveys or public opinion. It certainly happens in other areas of life. For example, in the writing and publishing...

My thoughts too. It's dross.

Wading into the fatigue versus PEM thing. Fatigue is a wholly unhelpful description (for me) because it's present in scores of chronic illnesses. It doesn't distinguish ME from other illnesses. Besides, I'm fatigued every day, and can live with that. But PEM is insidious. It means I can never...

Absolutely. Central and peripheral mechanisms seem to be at work. CSS attributes anything peripheral to the central dysfunction, so it ignores metabolic findings, for instance.

I have no problem with clinics trying to address neuroinflammation and activated glial cells in the brain. But CSS dumbs down and co-opts these findings for a BPS agenda.

Same. No benefit but lots of fishy burps! The evidence base is quite muddled too--it can't decide whether we need more omega 3 (fish oils) or omega 6 (evening primrose oil). Yet more of one usually means you absorb less of the other, if I'm remembering correctly. You can get omega 3 from...

Most UK doctors don't test for things like EBV. They don't seem to want to give antivirals either. You're usually told to rest up and drink plenty of fluids, then sent on your way again.

Have you considered sending them the Geraghty study of reported harms?

Why is this one getting so few compared to the MEA's petition? Petition fatigue? Or is it just not worded as well?

So much of this chimes with me.

Can anyone find this in any trial registries? I'm trying to find out what the hypothesis/proposal is?

There is. I'm on the board for a charity. I have to disclose anything that might count. My mum is a local councillor, she has to declare everything! There's a policy of 'whiter than white', because otherwise you bring disrepute to every person you work with. They don't care. Presumably because...

As always, it's overly simplistic. I think they're latching onto things like evidence of primed glial cells in the brain, and low level neuroinflammation, and mangling it with a biopsychosocial agenda. They expect to desensitize with rehabilitation, of course. What they fail to ignore is the...

I think cetirizine is now being touted as a useful secondary treatment alongside LDN by Dickson Pharmacy and the LDN Trust. They think this is because it may have remyelinating properties, although studies are needed to show this, in my opinion.

There is an objective reality here. He had things *other people* might consider COIs, so he should have declared them, regardless of whether he thought they were COIs.

There's an Oxford study comparing ME to bipolar. It's sponsored by the psychiatry department, but it is an MRS study, looking at lactate.

AfME weren't initially invited to sign and then didn't want to sign it late, IIRC. Convenient, you might think . . .

Read more: https://www.hindawi.com/journals/mi/2008/792428/ This is an old-ish study, bit has some interesting ideas about neurological conditions. Though it doesn't mention ME directly, three of the references used to support the argument deal with 'CFS'. The theory goes that interrupted VNs...

So basically, another study designed to let the authors do what they were going to do anyway.

Or doctors are kidding themselves that they're Jesus Christ of the Antidepressants! I've now read Dr Smith's rationale for ADs in ME, and it's a mess. Obviously disheartened by some contradictory findings about muscle dysfunction (which fly in the face of more recent CPET studies and others)...